New evidence has been highlighted by a team of researchers to suggest that Parkinson’s disease may have an infectious or autoimmune origin. The study was conducted by the NeuroGenetics Research Consortium, an international team of researchers led by Haydeh Payami, research scientist at the New York State Department of Health Wadsworth Center.

It is believed that results of this study will help the medical fraternity to take a fresh look at the possible role of infections, inflammation, and autoimmunity in Parkinson’s disease.

From Esciencenews.com:

People who take non-steroidal anti-inflammatory drugs (NSAIDs, such as ibuprofen) have a reduced risk of developing Parkinson’s disease, which also supports an immune-related mechanism. The protective effect of NSAIDs is not the same for everyone, likely because of genetic differences, and NSAIDs can have side effects. Pursuing the connection between Parkinson’s disease and inflammation, especially in the context of variable genetic makeup, may lead to better, more selective drugs for treating Parkinson’s disease.

Consortium leaders recognize the study would not have been possible without the precious help from volunteer patients. “This type of research could not be done if it weren’t for the willing and dedicated individuals who volunteer as research subjects,” said Payami, who acknowledged that some study subjects participated for nearly two decades.

The clinical directors for the study were Dr. Cyrus Zabetian, associate professor of neurology at the University of Washington and VA Puget Sound Health Care System, Stewart Factor at Emory University, and John Nutt at Oregon Health and Sciences University.

Tags: ibuprofen, NSAIDs, Parkinson's disease, steroidal

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