According to a new research, prolonged pulmonary rehabilitation can prove out to be an effective option for ensuring a slowdown decline of lung function in COPD patients.

Study leader Dr David Stav, a pulmonary specialist from Tel Aviv University, Israel, said that 3 years of pulmonary rehab is effective since it offers innumerable benefits on the rate of FEV1 decline along with advantages of this treatment modality for patients.

From Pulsetoday.co.uk:

Their study is the first to show benefits of pulmonary rehabilitation for disease progression, with patients receiving the treatment having significantly better lung function than controls after three years.

Researchers randomised 80 patients with moderate to severe COPD to receive pulmonary rehabilitation or usual care, consisting of follow-up appointments with a pulmonary specialist every three months.

After three years, patients receiving pulmonary rehabilitation had a decline in FEV1 of 74ml, compared with 149ml in the control group.

The pulmonary rehabilitation group also had significantly improved exercise endurance, maximal sustained work limits and BMIs, compared with controls.

Study leader Dr David Stav, a pulmonary specialist from Tel Aviv University, Israel, said: ‘Three years of pulmonary rehabilitation has an important beneficial impact on the rate of FEV1 decline, in addition to previously reported advantages of this treatment modality for COPD patients.’

The results - published online by the journal BMC Pulmonary Medicine - are a boost for the Department of Health’s forthcoming clinical strategy for COPD, due to be released later this year, which is expected to focus on the patchy provision of pulmonary rehabilitation.

These findings are expected to offer a new paradigm when it comes to finding effective treatment ways to cure COPD patients.

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According to a new study conducted by researchers from the Ohio State University Medical Center, a substance created by the immune cells play a pivotal role in disease progression in animals, closely mimicking multiple sclerosis. The study found out that blockage of this molecule known as macrophage migration inhibitory factor (MIF) can effectively prevent disease progression.

Though MIF may be considered of less importance for MS initiation, it is imperative for the progression of multiple sclerosis, as per principal investigator Caroline C. Whitacre, Professor of molecular virology, immunology and medical genetics.

From News-Medical.Net:

“Our results suggest that MIF may be less important for initiating MS, but that it may be necessary for MS progression,” says principal investigator Caroline C. Whitacre, professor of molecular virology, immunology and medical genetics.

“These findings indicate that in the future we can perhaps use MIF levels to predict the onset of a relapse. But more importantly, perhaps this study will lead to drugs that can halt the course of MS by blocking the action of MIF.”

The findings clearly suggested that MIF levels can be used for predicting the onset of a relapse. Most importantly, the study is believed to create new roadways for halting the course of MS by blocking MIF activities.

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