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iSteroids Newsletter

Issue # 11

Dr.Jekyll & Mr.Hyde

Body Transformation From Both Sides of the Force

Dr. Jekyll and Mr.Hyde: Body transformation from both sides of the force is a unique book because it deals with both regular and enhanced training. The natural athlete will find all the
tools (as well as a yearly training plan) training and nutrition-wise to transform his body into a lean and muscular work of art while the devoted bodybuilder will be able to learn a few new tricks regarding the very intensive training required to transform his body in a muscular machine.

Lean Mass Cycle-the evolution of the Bulking Cycle! By Kevin G. (aka Bajan Bastard) Ah the bulking cycle….that special time for the bodybuilder, when the goal is to pack on as much mass as possible in the shortest amount of time. It also gives you, this big guy who so into fitness the perfect excuse as to why you’re in a fast food joint with your face covered in pizza sauce and chicken grease. “But I’m bulking!”-Says the Lee Priest in all of us.

So what truly defines a bulking cycle? Let’s define exactly what a bulking cycle actually is, shall we?

When asked, most AAS users will tell you a “bulking cycle” is used to acquire maximum amount of size and mass in a certain time period, also when asked the typical AAS user will also choose a select few out of the dozens of anabolic steroids available to them. So what are these drugs that are the usual suspects in the bulking cycle and why? Well, first off, some of the obvious choices are anabolic hormones that promote a rapid increase in overall mass. The first drug chosen almost all the time for the foundation of a bulk cycle is the great granddaddy of all steroid hormones, yeah you guessed it right, testosterone. Testosterone is the primary androgen in the male body and is a potent anabolic and androgen, it possess an androgenic/anabolic ratio of 100/100 and because it can aromatize (convert via the aromatize enzyme) into estrogen, water retention from the estrogen contributes to the overall size, volume and strength of the person who is fortunate enough to be using testosterone, during a bulking cycle a testosterone attached to a longer, slower acting ester is usually preferred, examples would include; Testosterone Enanthate, Testosterone Cypionate and testosterone Decanoate. Testosterone dosages can range anywhere from 300mg per week to 2000mg or in some cases per week.

The ever popular anabolic steroid, Deca Durabolin (Nandrolone Decanoate) is another steroid that is found in almost all cycles of this type. ‘Deca’ as it’s so affectionately known is basically testosterone without a carbon atom at the 19th position, this seemingly insignificant modification has a profound effect on the properties of the hormone however. First, Nandrolone is an excellent muscle builder, yet not very androgenic, so the (over) development of male sexual characteristics are not a huge concern to the user, it also has a low aromatization rate, roughly 20% of that of testosterone. Deca also ‘lubes’ the joints as fans of this hormone love to say. Deca indeed promotes collagen synthesis and bone mineral build-up(1-2) and help those who has troubled joints push that heavy iron during their bulk phase. It does have the undesirable trait of being a ‘progestin’, meaning it acts similarly to the female sex hormone progesterone (somewhat). In the case of Deca it actually binds to the progesterone receptor at about 20% the rate of actual progesterone. Sounds low but Deca does cause water retention in most, if not all people who use it. This may be because estrogenic effects are exacerbated by the presence of progesterone/progestins. Deca is most commonly dosed at 400mgs/week up too 1000 or more mg weekly.

The inclusion of an oral that causes a quick increase in muscle mass and strength is also a common part of a cycle of this type. Dianabol (methandrostenolone) the ‘Breakfast of Champions’ and Anadrol (Oxymetholone) also known as ‘A-bombs’ are the two most popular ‘bulking orals’ used by today’s bodybuilders.

Dianabol seems to be the preferred oral of the anabolic steroid coinsurer for bulking. Users appreciate the effects of this drug, which include ‘mass’ gains from increased intramuscular nitrogen retention, glycogen and protein synthesis(3-4), a feeling of well being and more ‘real’ muscle gains kept after cessation.(4-5) Like all steroids dianabol does have its drawbacks. Being an oral steroid its chemical structure makes it hard for the liver to metabolize the drug, called 17-alpha-alkalation (17AA), this modification increases the ‘liver-toxicity’. A general rule of thumb is to use an oral (17AA) drug for no more than 4-6 weeks, this greatly depends no the drug in question because some orals are more toxic than others. Unfortunately the liver toxic effects of 17AA orals have been greatly exaggerated, use orals for much longer time periods, yet it is still best to err on the side of caution. Dianabol can also aromatize into estrogen, so water retention, high blood pressure, and gynecomastia (gyno, bitch-tits) are sometimes reported.

20-50mg per day of dianabol for 4-6 weeks is the recommended dose for most bodybuilders.

Anadrol (a.k.a. A-bombs) is one of the most powerful oral anabolic agents readily available. It is capable of increasing an individual’s overall mass and strength exponentially in a matter of weeks. If you’re seeking a drug for seer ‘bulk’ A-bombs are what you’re looking for. Anadrol is a strange drug in the sense that although it does not convert to estrogen, it causes a great deal of estrogenic side effects, high blood pressure, water retention, gyno are commonly reported from Anadrol usage. A-bombs are notorious for totally killing the appetite of some individuals who used the drug, accompanied by nausea and upset stomach. Not exactly a good feeling during a time when plenty of extra calories must be consumed on a daily basis. Anadrol is typically used 50-200mg daily for 3-4 weeks for its effects until the other slow acting injectables ‘kick in’. Steroid researcher Anthony Roberts recently discovered that 100mg per day of anadrol was the optimal dose for the particular compound with decreased return and increase side effects as the drug was admin istered at a higher dose.(4)

Here is an example of a bulking cycle:

Testosterone Enanthate 600mg weekly 14 weeks

Nandrolone Decanoate 400mg weekly 12 weeks

Methandrostenolone 30mg daily 1st 4-6 weeks.

With the advent of the powder trade and UGL’s (Underground Labs, for those not up with the current jargon) willing to produce high quality product at reasonable prices and research companies providing once expensive, hard to find ancillaries, the traditional bulking cycle has be modified with the addition of new or formerly obscure anabolic hormones and esters. The bulking cycle entered a state of change and emerged, evolved and refined as the ‘Lean Mass Cycle’ (hereafter to be referred to as the “L.M.C” or just LMC).

As the name suggest the LMC is a cycle where the user wants to gain weight but not at all costs. The main goal is muscle yes, but all the while minimizing excessive fat and water gain. LMCs typically resembles ‘cutting cycles’ in terms of some drug choices and ancillary use, only thing that is different is the dosages used may be higher. Lets face it, nobody likes to get totally fat in the off season, even if it’s fun to be able to eat anything you want. The LMC consists primarily of anabolic steroids that have a low estrogen conversion or no estrogenic properties or ones that do not convert to estrogen at all. They also utilize short estered variants of other popular drugs. Personally I never liked or understood the concept of the bulking cycle, why someone would want to look like a blimp on cycle as always beyond me. Furthermore getting fat on cycle was, in my honest opinion, a very silly idea. Just to do what? Having to run a cutting cycle after the bulking one? Nonsense! A LMC is the much better option in my eyes. Ok, yeah, you’re sick of reading about what I think right? Right. So let’s get into the meat of the matter; exactly which drugs are used during a LMC.

LMC like almost all other good cycles have a base of testosterone but this time around testosterone propionate is the perennial choice, if the user is uncomfortable with the sometimes painful injections and every day at that, a testosterone with a longer ester may be user along side an aromatize inhibitor to prevent water retention and fat build-up. Another top favorite is the mighty Trenbolone… oh man Trenbolone. I actually used this drug at a mere 75mgs every other day (called “EOD” in steroid jargon) alongside 500mgs of Testosterone Enanthate for my 1st cycle…and even though I only used it for 6 weeks, I’ve been in love with it ever since.

Anyway, to cap off the LMC the addition of a DHT derivative is always welcomed. Stanozolol (Winstrol) or Drostanolone (Masteron) are the ones usually chosen to fit this slot. To be honest unlike a bulking cycle almost all androgens are useful in a LMC. Remember, typically bulking cycles are dependant on total mgs/week, while cutting cycles are more dependant on actual compounds used (although diet will really be the deciding factor in either of these types of cycles). So, clearly, I cannot possibly list all of the drugs you could ever use in a LMC, and their properties as well. However I will list the most the popular and easily available drugs used to construct a LMC and for only 50 east payment of $19.99 I’ll add some sample cycles! Ok I’m done, jokes where never my forte anyway. Here is that list I mentioned:










These drugs are relatively easy to acquire and cost effective for the job they were selected for….well except for Methenolone and Oxandrolone save those for when your kid is of out of Flintstone vitamins.

So here is a sample cycle incorporating a few of the aforementioned drugs
Testosterone Propionate 100mg daily 10 weeks
Trenbolone Acetate 75mg daily 8 weeks
Drostanolone Propionate 50mg daily 8 weeks
Chlorodehydromethyltestosterone 50mg daily 1st or last 6 weeks

Masteron as an Anti-Estrogen

I know it must seem like I sit around all day trying to find new uses for old drugs, but in this case, nothing could be further than the truth. Before I get into how and why you can use Masteron as an Anti-Estrogen, I’ll tell you a bit about where this idea came from, and why I’m telling you about it. And yes, this works in real life, not just on paper - I’ve used it and seen it used for this purpose successfully by several athletes.

A few years ago, I wrote my first piece on Masteron (Drostanolone Propionate), and discovered what its clinical use actually was: Reduction of breast cancer tumors, and as hormonal treatment of breast cancer. Well…the long version of that is that Masteron is an androgenic, anabolic steroid, used as an agent used to prevent or inhibit the growth of cancerous tumors.

Then, in one of those weird "duh" moments, I realized that gynocomastia, mastectomy, and Masteron all had that similar word root. You’d think that having an English degree would have helped me notice this fact sooner…anyway, I wrote the profile and didn’t think much about it anymore. I was then contacted by the owner of an underground lab, and asked why Masteron was always produced with a propionate ester, and whether it would be ok with a longer ester. This began another long period of research for me into Masteron. Well, I found out that Masteron would be fine with a longer ester, but I actually had a chance to test it out with that particular ester before it hit the market...I was still standing after 3 weeks on it, so it was produced en masse (as a side note that particular Underground Lab still produces it and it’s one of their better selling products).

So here I was with all of this research on Masteron and nothing to do with it. Well, after I took another look at the compound, a couple of things struck me. The first that struck me is that Masteron is made for women! Yeah…go back and read that again if you have to. Masteron is one of the few steroids that were actually created with women in mind, not men, and it’s the one that most people tell women to avoid! And the other thing that I noticed right away was that it is used for treatment of breast cancer. In particular, it’s used for the treatment of estrogen dependant breast tumors. By now, I’m sure you see where I’m going with this…Nolvadex is used clinically for this same purpose, as is Arimidex, Femera, Aromasin (a steroidal Aromatase Inhibitor), and Teslac (a steroid, technically). That’s some good company to be in, if you’re a steroid. But interestingly, Teslac is actually a steroid also, and Aromasin is a Steroidal Aromatase Inhibitor. So why can’t a "real" steroid do the same job at preventing breast cancer? Well, the answer is that it can!

To understand why Masteron can be used as an anti-estrogen, first we need to know that it’s derived from DHT. Why is this important?

This is important because DHT directly inhibits estrogenic activity on tissues. It is possible that it does this, possibly by acting as a competitive antagonist to the estrogen receptor or by decreasing estrogen receptor binding. Either way, it has multiple hypothesized mechanisms of action in some tissues. It has also been hypothesized that DHT actually suppresses estrogen’s effects not by inhibition of synthesis of estrogen receptor, but by (get ready…big words coming up) decreasing estrogen-induced RNA transcription at some point after the actual estrogen receptor binding has occurred. This means, in much simpler terms, that the estrogen gets to the receptor, but just doesn’t do its job (1). This means you can take steroids that convert to estrogen (called aromatizable steroids) and not worry about that estrogen possibly making you retain water, gain fat, or watch "Desperate Housewives." Also, this could mean that the antiestrogenic effect of DHT is mediated by an androgen receptor mediated mechanism. In fact, DHT has been shown to prevent the estrogen-dependent augmentation of the progesterone receptor in human breast cancer cells. And, not to be redundant, but it’s important to remember that virtually all of the anti-estrogens we use to control gyno and water retention are also used to treat breast cancer. So, now we know have observed that androgens are capable of inhibiting both the estrogenic induction and the ongoing stimulation of PRc synthesis, but have no apparent effect upon basal concentrations of this receptor. Dihydrotestosterone (DHT) demonstrates a very high degree of inhibition of estrogen in human breast cancer cells. (2). But it’s not just DHT that does this; its metabolites have been shown to inhibit aromatization itself; DHT, androsterone, and 5alpha-androstandione are all potent inhibitors of the formation of estrone from androstenedione. In fact, it's so potent at reducing estrogen that transdermal DHT gel applied to the affected area has been used to treat gynocomastia (3). DHT is such a potent anti-estrogen that it been even been used to increase height in children with short stature, and since it’s been determined that this increase is not due to GH-mediated effects, it was strongly suggested that DHT’s anti-estrogenic effects are the mechanism by which it can increase height (4) Of course, I suspect I don’t need to tell you that DHT is structurally incapable or converting to estrogen…

So all of this tells us that DHT will certainly have beneficial effects on keeping our estrogen in check, but what about Masteron? Can it be used as effectively? Well, let’s take a look at what Masteron actually is, relative to DHT. But before we can do that, I think a quick explanation of DHT is in order first. Don’t worry; I’ll make it as brief and painless as possible.

DHT is actually the result of testosterone interacting with the 5alpha-reductase (5a-R) enzyme. This enzyme is present in the scalp, prostate, external genitalia, and other places. As far as I can see, it apparently exists for the sole purpose of converting a steroid with a double bond between carbon 4 and carbon 5 to one with a single bond between them, and subsequently adding a hydrogen atom to each carbon. This process is called (of course) 5alpha-reduction.




(Testosterone) (Dihydrotestosterone)
So now we know how testosterone becomes Dihydrotestosterone. And everything would be great if this is the only thing that happened to our good old friend testosterone, because as you may already know, DHT is a far more potent androgen than testosterone. But, unfortunately, this is not the end of the story, because DHT is largely deactivated by the enzyme 3-alpha Hydroxysteroid Dehydrogenase (3bHSD), which is mainly present in skeletal muscle.

For our purposes here, we’re only going to be concerned with one particular action of this enzyme. It can either converts a steroid with a keto group on position 3 of the steroid to one with a hydroxy group in that position, thus converting DHT is to androstanediol. This conversion is part of reason DHT alone has not proven to be a very effective muscle builder, as androstanediol is not going to be very anabolic at all. If you look off to the left of the following molecular diagram, and compare it to the one above for DHT, you’ll notice that the "O" (oxygen) has been replaced with an "HO" (hydrogen + oxygen) at the third position:


3bHSD is present all over the body (as is 5a-R, for the most part), but is found in especially high concentrations in the scalp and prostate, and it’s even possible that its actions on DHT will exacerbate male pattern baldness in the former tissue. Also, it’s worth noting that DHT is the androgen responsible for development of external genitalia. This is most likely the reason that women experience a temporary clitoral hypertrophy when they use the often recommended steroids (Primobolan, Anavar, Winstrol, etc…) in excessive doses. In an interesting aside, I find it really interesting that the most typical steroids recommended are the most likely to cause clitoral enlargement and other possible androgenic effects. But on the bright side, in my experience with female athletes, that first effect is most welcome...actually, topical DHT can even be used to treat Microphalia (extremely tiny genitalia) in males (5). This last fact, if you’ve ever wondered, is the type of information discussed behind closed doors by of owners and staff of "private/invite-only" anabolic steroid boards and forums…for obvious reasons…

Ok, so now you know what DHT is, where it comes from, what it can do, and why it’s not a particularly potent anabolic when used alone. Here’s what Masteron is, relative to its parent compound, DHT. Masteron is an injectable steroid that is simply the DHT molecule which has been altered to be 2alpha-Methyl-DHT…you can see this modification by comparing the DHT molecule above with the following Masteron one, and paying special attention to the left hand side again, and the "H3C" modification:

(Masteron, aka Drostanolone Propionate)

This 2-alpha-methyl alteration makes it much more potent anabolic, although it’s still only about 60% as anabolic as testosterone and a quarter as androgenic. I’m going to speculate that these ratings make it not the most potent anabolic in the world, but its anti estrogenic effects plus its ability to increase aggression make it a very nice pre-contest addition. This is also where we get the absurd rumor that Masteron won’t do anything for you unless you’re already at a very low body-fat percentage. This is not true at all. No matter what body-fat percentage you’re at going to get a nice anti-estrogenic effect from Masteron, as well as some nice aggression and strength in the gym - the former and latter are both known as "non-genomic" effects, and are a result of the strong Central Nervous System stimulatory effects of Masteron, which is very common with DHT derived steroids. Basically, if you’re fat, and you take something that increases aggression and has anti-estrogenic effects (Halotestin and Arimidex, lets say), you wouldn’t expect to get huge and ripped. It’s the same thing with Masteron. Now, what if you add in Arimidex and Halotestin to a pre-contest cycle, you’ll get harder and look better. That’s exactly what’ll happen if you add Masteron into a Pre-contest cycle. It’s not that you have to be at some random body-fat percentage to get results from it, but you’ll need to be at that lower body-fat percentage to "see" those results. Again, if you’re fat and take Halo and Arimidex, you aren’t going to look much better…think of Masteron in similar terms, but it won’t work as well for aggression as Halotestin, and won’t be as good for combating estrogen as Arimidex. Gauged against either one of them alone, Masteron will likely make you look much harder and lift more weight. But if you are looking to do a low dosage cycle with a minimal amount of compounds in it, a simple Testosterone (propionate) and Masteron cycle may be exactly what you are looking for. On a personal note, that is a cycle that I use very frequently, at about 100mgs of each, shot every other day.

But has Masteron actually lived up to my claims for being an anti-estrogen? Yes. From 1968 to 1972, a decent sized study was conducted on Masteron, in a group of premenopausal women with breast cancer. About a third responded well to Masteron (6). This is because of its anti-estrogenic effects, clearly- though it doesn’t perform as well as Arimidex, Letrozole, or Aromasin. If you’re not running huge amounts of aromatizable steroids, this is a very good choice to add into your cycle. If you’re doing large amounts of those compounds, then you need to use a traditional anti-estrogen as your ancillary compound of choice. But if you’re running well under a gram of aromatizable steroids, Masteron will likely be all the anti-estrogen you need. This number comes from my person experience, as well as others I’ve interviewed.

Now, as a bit of an addendum, I’d like to address the use of Masteron in women. Lets get this straight: Masteron was developed for women. Okay? Got me? If you’ ve been paying attention up to this point, you already know that Masteron is intended for females and is derived from the same root (DHT) as most other steroids commonly used and recommended for female athletes (Primobolan, Anavar, Winstrol, etc…are all derived from DHT). And, another shocking fact is that Masteron has a lower androgenic rating than almost every other commonly recommended steroid used by female athletes. Anavar has a rating of 24 compared to oral testosterone and Masteron has a rating of 25 compared to testosterone, expressed as a percent (so yes that means 24% and 25% respectively).

Basically, Masteron works as a hormonal therapy for breast cancer and has been shown to be a useful and safe agent for females of all age groups, even though it may appear to be less effective then other possible therapies in postmenopausal patients (6). It is, therefore, very safe for women. Masteron is certainly no less safe than Anavar or Primobolan for women, as long as it’s used with something resembling a degree of respect and intelligence.

My recommendations for female use of this compound would be to start between 10-25mgs every third day, and increase dosages from there if no side effects are experienced. At those dosages, I suspect no side effects would be experienced, and I’d be comfortable saying none will be experienced up through 20mgs, injected every other day.

So there you have it. A totally new way to look at an old friend- Masteron- it’s useful as an anti-estrogen as well as an anabolic, and can certainly be safely used by both Men as well as women.


J Steroid Biochem. 1983 Oct;19(4):1513-20.

Journal of Clinical Endocrinology & Metabolism, Vol 53, 836-842, Copyright © 1981 by Endocrine Society

Successful percutaneous dihydrotestosterone treatment of gynecomastia occurring during highly active antiretroviral therapy: four cases and a review of the literature. Clin Infect Dis. 2001 Sep 15;33(6):891-3. Epub 2001 Aug 10.

J Clin Endocrinol Metab. 1993 Apr;76(4):996-1001.

Baillieres Clin Endocrinol Metab. 1998 Oct;12(3):501-6

Hormonal therapy of breast cancer with special reference to Masteron therapy. Bennett MB, Helman P, Palmer P PMID: 1242823).

Targex - The Ultimate Topical Fat Burner!



Glycyrrhetinic acid is the primary ingredient in Targex. It is actually found in licorice, and when applied to the skin, can reduce the thickness of subcutaneous fat. In this case, the mechanism of action is mediated by the catabolic hormone known as cortisol. This hormone is involved in both the distribution as well as the deposition of fat.Cortisol, in turn is regulated by the activity of an enzyme which Glycyrrhetinic acid (the active ingredient in Targex) blocks. This reduces the ability of cortisol to regulate fat cells, resulting in an overall loss of bodyfat to the area the cream is applied to.

In a clinical study, the effect of topical Glycyrrhetinic cream was evaluated. In this study, measurements of thigh fat, before and after 1 month of treatment with the cream were taken. In both areas (circumference and the fat layer thickness) the thighs receiving the cream had significantly lower levels of fat.

Hence, this type of cream would appear to be a potent lipolytic (fat burning) agent. Also, it is important to note that this compound is very safe, as the full study mentioned previously states that that there was absolutely no change in plasma cortisol, blood pressure, or aldosterone.

The primary ingredient in that study (Glycyrrhetinic acid) is also the primary ingredient found in Targex; this is actually the brand name of the original version of these types of products. Typically it is applied to specific sites, to produce localized fat-loss wherever it is applied. The typical Targex user has experienced significant reductions in body fat when it was applied to site-specific areas, and was used in combination with a proper diet, training, and cardio.


Glycyrrhetinic acid, the active principle of licorice, can reduce the thickness of subcutaneous thigh fat through topical application.

Armanini D, Nacamulli D, Francini-Pesenti F, Battagin G, Ragazzi E, Fiore C.

Department of Medical and Surgical Sciences-Endocrinology, University of Padua, Via Ospedale 105, 35100 Padua, Italy.

Cortisol is involved in the distribution and deposition of fat, and its action is regulated by the activity of 11beta-hydroxysteroid dehydrogenase. Glycyrrhetinic acid, the active principle of licorice root, blocks 11beta-hydroxysteroid dehydrogenase type 1, thus reducing the availability of cortisol at the level of adipocytes. We evaluated the effect of topical application of a cream containing glycyrrhetinic acid in the thickness of fat at the level of the thigh. Eighteen healthy women (age range 20-33 years) with normal BMI were randomly allocated to treatment, at the level of the dominant thigh, with a cream containing 2.5% glycyrrhetinic acid (n=9) or with a placebo cream containing the excipients alone (n=9). Before and after 1 month of treatment both the circumference and the thickness of the superficial fat layer of the thighs (by ultrasound analysis) were measured. The circumference and the thickness of the superficial fat layer were significantly reduced in comparison to the controlateral untreated thigh and to control subjects treated with the placebo cream. No changes were observed in blood pressure, plasma renin activity, plasma aldosterone or cortisol. The effect of glycyrrhetinic acid on the thickness of subcutaneous fat was likely related to a block of 11beta-hydroxysteroid dehydrogenase type 1 at the level of fat cells; therefore, glycyrrhetinic acid could be effectively used in the reduction of unwanted local fat accumulation.

If you read the original abstract you will note that ...

"No changes were observed in blood pressure, plasma renin activity, plasma aldosterone or cortisol",

There was no change in plasma cortisol; cortisol was lowered only in the fat cells, this is telling us that little if any of the compound was absorbed systemically or we would see a drop in whole body, i.e. plasma cortisol.



Apply 1 to 2 pumps ( one pump = one CC ) applied TWICE daily in desired areas and rub vigorously until almost vanished into the skin of area applied, though this lotion is greasy of nature is necessary for it to work locally.

The area must be clean and free of hair.

This lotion will NOT stain clothes.

There are some studies that claim Licorice extract may increase levels of the hormone adolsterone if there is a systemic uptake BUT our unique TDS formual DOES NOT allow this to happen and TargeX works on the site applied.



So I have some problem areas where I store more fat or simply I got some flabby stubborn fat like for example in my oblique , inner thighs and lower pecs , my diet and cardio has been in check but still I need more help!

Well, TargeX will not shift your body into a calorie burning machine like other compound do ie. Clenbuterol , T3 , Ephedra , etc...but BY BLOCKING CORTISOL AT THE CELLULAR LEVEL AND PREVENT IT IT FROM STORING FAT WHEN THIS HORMONE "CORTISOL" IS AT PEAK ( BEFORE RETIRING, UPON AWAKENING AND POST-WORKOUT ) Of course add a good diet and cardio and you'll see visible results in the areas applied.


Ok, so I got a 4oz bottle of TargeX and it contains 120cc of 40mgs of G.Acid 98% purity per CC enough for a 2 week cycle which is what i recommend as our wonderful bodies will fight it, after all cortisol hormone is a necessary evil so cycling 2 weeks on - 2 weeks off one area at a time is best !

Patience and common sense is needed here, please allow me to explain why and how:

I want to decrease the stubborn fat I got on my oblique both left and right so :

Before I go to bed I apply 2ccs of TargeX on my right oblique then rub vigorously until vanished ( I like using a hair blowdrier in Med tempetature to help dry area faster )

Then I apply another 2ccs on my left oblique and rub again until vanished and absorbed.

Next day when I wake up I repeat procedure again.

***Make sure the area you apply TargeX to is free of hair and is clean.

Using TargeX this way is safe and ONE 4oz bottle should be enough for a 2 week cycle and may have some extra.

Ok, now you see some improvement in the areas you applied to but you need more treatment so you start again another 2 week cycle after at least 1 week off TargeX, if you are happy with your results then move on to another area you desired to reduce stubborn subcutaneous fat. This time it may the lower pecs so you do exactly as you did the first time when you applied to your oblique.

***Please keep in mind this is not miracle drug ( well almost ) and that the area you worked first may need some more treatment but you need to do your part as far as diet and cardio, this is true in all products that help reduce body fat, do not tell me is not true cause I tried them all and when my diet was sloppy I made no progress.

***Work one area at a time, I mean for example again oblique ( left and right ), lower pecs ( left and right ) inner thighs ( left and right) and so on

***I recommend using an over the counter dietary supplement that reduces excess estrogen , is my belief that the presence of excess estrogen makes that stubborn fat hang on to those problem areas. This is just my own opinion based on personal experiences.

So there you have it. So the next time you decide to ‘bulk’ you’ll know have much more options than you previously thought.
  1. Metabolism. 1990 Nov;39(11):1167-92.
  2. Effects of nandrolone decanoate on bone mineral content R, Righi GA, Turchetti V, Vattimo A.
  3. Med sci. sprots1979 Summer;11(2):160-3.
  4. Anabolic Steroids, Ultimate Research Guide. Anthony Roberts & Brian Clapp, Anabolic Books LLC, Montgomery TX, 2005.
  5. Various steroid user reports.
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