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Dr. Jekyll and Mr.Hyde: Body transformation from both sides of the
force is a unique book because it deals with both regular and enhanced
training. The natural athlete will find all the
tools (as well as a yearly training plan) training and nutrition-wise
to transform his body into a lean and muscular work of art while the
devoted bodybuilder will be able to learn a few new tricks regarding
the very intensive training required to transform his body in a
Lean Mass Cycle-the evolution of the Bulking Cycle! By Kevin
Bajan Bastard) Ah the bulking cycle….that special time for the
bodybuilder, when the goal is to pack on as much mass as possible in
the shortest amount of time. It also gives you, this big guy who so
into fitness the perfect excuse as to why you’re in a fast food joint
with your face covered in pizza sauce and chicken grease. “But I’m
bulking!”-Says the Lee Priest in all of us.
So what truly defines a bulking cycle? Let’s define exactly what a
bulking cycle actually is, shall we?
When asked, most AAS users will tell you a “bulking cycle” is used to
acquire maximum amount of size and mass in a certain time period, also
when asked the typical AAS user will also choose a select few out of
the dozens of anabolic steroids available to them. So what are these
drugs that are the usual suspects in the bulking cycle and why? Well,
first off, some of the obvious choices are anabolic hormones that
promote a rapid increase in overall mass. The first drug chosen almost
all the time for the foundation of a
bulk cycle is the great granddaddy of all steroid hormones, yeah you
guessed it right, testosterone. Testosterone is the primary androgen in
the male body and is a potent anabolic and androgen, it possess an
androgenic/anabolic ratio of 100/100 and because it can aromatize
(convert via the aromatize enzyme) into estrogen, water retention from
the estrogen contributes to the overall size,
volume and strength of the person who is fortunate enough to be using
testosterone, during a bulking cycle a testosterone attached to a
longer, slower acting ester is usually preferred, examples would
include; Testosterone Enanthate, Testosterone Cypionate and
testosterone Decanoate. Testosterone dosages can range anywhere from
300mg per week to 2000mg or in some cases per week.
The ever popular anabolic steroid, Deca Durabolin (Nandrolone
Decanoate) is another steroid that is found in almost all cycles of
this type. ‘Deca’ as it’s so affectionately known is basically
testosterone without a carbon atom at the 19th
position, this seemingly insignificant modification has a profound
effect on the properties of the hormone however.
First, Nandrolone is an excellent muscle builder, yet not very
androgenic, so the (over) development of male sexual characteristics
are not a huge concern to the user, it also has a low aromatization
rate, roughly 20% of that of testosterone. Deca also ‘lubes’ the joints
as fans of this hormone love to say. Deca indeed promotes collagen
synthesis and bone mineral build-up(1-2) and help
those who has troubled joints push that heavy iron during their bulk
phase. It does have the undesirable trait of being a ‘progestin’,
meaning it acts similarly to the female sex hormone progesterone
(somewhat). In the case of Deca it actually binds to the progesterone
receptor at about 20% the rate of actual progesterone. Sounds low but
Deca does cause water retention in most,
if not all people who use it. This may be because estrogenic effects
are exacerbated by the presence of progesterone/progestins. Deca is
most commonly dosed at 400mgs/week up too 1000 or more mg weekly.
The inclusion of an oral that causes a quick increase in muscle mass
and strength is also a common part of a cycle of this type.
Dianabol (methandrostenolone) the ‘Breakfast of Champions’ and Anadrol
(Oxymetholone) also known as ‘A-bombs’ are the two most popular
‘bulking orals’ used by today’s bodybuilders.
Dianabol seems to be the preferred oral of the anabolic steroid
coinsurer for bulking. Users appreciate the effects of this drug, which
include ‘mass’ gains from increased intramuscular nitrogen retention,
glycogen and protein synthesis(3-4), a feeling of well being and more
‘real’ muscle gains kept after cessation.(4-5) Like all steroids
dianabol does have its drawbacks. Being an oral steroid its chemical
structure makes it hard for the liver to metabolize the drug, called
17-alpha-alkalation (17AA), this
modification increases the ‘liver-toxicity’. A general rule of thumb is
to use an oral (17AA) drug for no more than 4-6 weeks, this greatly
depends no the drug in question because some orals are more toxic than
others. Unfortunately the liver toxic effects of 17AA orals have been
greatly exaggerated, use orals for much longer time periods, yet it is
still best to err on the side of caution. Dianabol can also aromatize
into estrogen, so water retention, high blood pressure, and
gynecomastia (gyno, bitch-tits) are sometimes reported.
20-50mg per day of dianabol for 4-6 weeks is the recommended dose for
Anadrol (a.k.a. A-bombs) is one of the most powerful oral anabolic
agents readily available. It is capable of increasing an individual’s
overall mass and strength exponentially in a matter of weeks. If you’re
seeking a drug for seer ‘bulk’ A-bombs are what you’re looking for.
Anadrol is a strange drug in the sense that although it does not
convert to estrogen, it
causes a great deal of estrogenic side effects, high blood pressure,
water retention, gyno are commonly reported from Anadrol usage. A-bombs
are notorious for totally killing the appetite of some individuals who
used the drug, accompanied by nausea and upset stomach. Not exactly a
good feeling during a time when plenty of extra calories must be
consumed on a daily basis. Anadrol is typically used
50-200mg daily for 3-4 weeks for its effects until the other slow
acting injectables ‘kick in’. Steroid researcher Anthony Roberts
recently discovered that 100mg per day of anadrol was the optimal dose
for the particular compound with decreased return
and increase side effects as the drug was admin istered at a higher
Here is an example of a bulking cycle:
Testosterone Enanthate 600mg weekly 14 weeks
Nandrolone Decanoate 400mg weekly 12 weeks
Methandrostenolone 30mg daily 1st 4-6 weeks.
With the advent of the powder trade and UGL’s (Underground Labs, for
those not up with the current jargon) willing to produce high
quality product at reasonable prices and research companies providing
once expensive, hard to find ancillaries, the traditional bulking cycle
has be modified with the addition of new or formerly obscure anabolic
hormones and esters. The bulking cycle entered a state of change and
emerged, evolved and refined as the ‘Lean Mass Cycle’ (hereafter to be
referred to as the “L.M.C” or just LMC).
As the name suggest the LMC is a cycle where the user wants to gain
weight but not at all costs. The main goal is muscle yes, but
all the while minimizing excessive fat and water gain. LMCs typically
resembles ‘cutting cycles’ in terms of some drug choices and ancillary
use, only thing that is different is the dosages used may be higher.
Lets face it, nobody likes to get totally fat in the off season, even
if it’s fun to be able to eat anything you want. The LMC consists
primarily of anabolic steroids that
have a low estrogen conversion or no estrogenic properties or ones that
do not convert to estrogen at all. They also utilize short estered
variants of other popular drugs. Personally I never liked or understood
the concept of the bulking cycle, why someone would want to look like a
blimp on cycle as always beyond me. Furthermore getting fat on cycle
was, in my honest opinion, a very silly idea.
Just to do what? Having to run a cutting cycle after the bulking one?
Nonsense! A LMC is the much better option in my eyes. Ok, yeah, you’re
sick of reading about what I think right? Right. So let’s get into the
meat of the matter; exactly which drugs are used during a LMC.
LMC like almost all other good cycles have a base of testosterone but
this time around testosterone propionate is the perennial
choice, if the user is uncomfortable with the sometimes painful
injections and every day at that, a testosterone with a longer ester
may be user along side an aromatize inhibitor to prevent water
retention and fat build-up. Another top favorite is the mighty
Trenbolone… oh man Trenbolone. I actually used this drug at a mere
75mgs every other day (called “EOD” in steroid jargon) alongside 500mgs
of Testosterone Enanthate for my 1st cycle…and
even though I only used it for 6 weeks, I’ve been in love with it ever
Anyway, to cap off the LMC the addition of a DHT derivative is always
welcomed. Stanozolol (Winstrol) or Drostanolone (Masteron) are
the ones usually chosen to fit this slot. To be honest unlike a bulking
cycle almost all androgens are useful in a LMC. Remember, typically
bulking cycles are dependant on total mgs/week, while cutting cycles
are more dependant on actual compounds used (although diet will really
be the deciding factor in either of these types of cycles). So,
clearly, I cannot possibly list all of the drugs you could ever use in
a LMC, and their properties as well. However I will list the most the
popular and easily available drugs used to construct a LMC and for only
50 east payment of $19.99 I’ll add some sample cycles! Ok I’m done,
jokes where never my forte anyway. Here is that list I mentioned:
These drugs are relatively easy to acquire and cost effective for the
job they were selected for….well except for Methenolone and Oxandrolone
save those for when your kid is of out of Flintstone vitamins.
So here is a sample cycle incorporating a few of the aforementioned
100mg daily 10 weeks
75mg daily 8 weeks
50mg daily 8 weeks
50mg daily 1st or last 6 weeks
Masteron as an Anti-Estrogen
I know it must seem like I sit around all day trying to find new uses
for old drugs, but in this case, nothing could be further
than the truth. Before I get into how and why you can use Masteron as
an Anti-Estrogen, I’ll tell you a bit about where this idea
came from, and why I’m telling you about it. And yes, this works in
real life, not just on paper - I’ve used it and seen
it used for this purpose successfully by several athletes.
A few years ago, I wrote my first piece on Masteron (Drostanolone
Propionate), and discovered what its clinical use actually was:
Reduction of breast cancer tumors, and as hormonal treatment of breast
cancer. Well…the long version of that is that Masteron is an
androgenic, anabolic steroid, used as an agent used to prevent or
inhibit the growth of cancerous tumors.
Then, in one of those weird "duh" moments, I realized that
gynocomastia, mastectomy, and Masteron all had that similar word root.
You’d think that having an English degree would have helped me notice
this fact sooner…anyway, I wrote the profile and
didn’t think much about it anymore. I was then contacted by the owner
of an underground lab, and asked why Masteron was always
produced with a propionate ester, and whether it would be ok with a
longer ester. This began another long period of research for me into
Masteron. Well, I found out that Masteron would be fine with a longer
ester, but I actually had a chance to test it out with that particular
ester before it hit the market...I was still standing after 3 weeks on
it, so it was produced en masse (as a side note that particular
Underground Lab still produces it and it’s one of their better selling
So here I was with all of this research on Masteron and nothing to do
with it. Well, after I took another look at the compound, a couple of
things struck me. The first that struck me is that Masteron is made for
women! Yeah…go back and read that again if you have to.
Masteron is one of the few steroids that were actually created with
women in mind, not men, and it’s the one that most people
tell women to avoid! And the other thing that I noticed right away was
that it is used for treatment of breast cancer. In particular,
it’s used for the treatment of estrogen dependant breast tumors. By
now, I’m sure you see where I’m going
with this…Nolvadex is used clinically for this same purpose, as is
Arimidex, Femera, Aromasin (a steroidal Aromatase Inhibitor), and
Teslac (a steroid, technically). That’s some good company to be in, if
you’re a steroid. But interestingly, Teslac is
actually a steroid also, and Aromasin is a Steroidal Aromatase
Inhibitor. So why can’t a "real" steroid do the same job at
preventing breast cancer? Well, the answer is that it can!
To understand why Masteron can be used as an anti-estrogen, first we
need to know that it’s derived from DHT. Why is this
This is important because DHT directly inhibits estrogenic activity on
tissues. It is possible that it does this, possibly by acting as a
competitive antagonist to the estrogen receptor or by decreasing
estrogen receptor binding. Either way, it has multiple hypothesized
mechanisms of action in some tissues. It has also been hypothesized
that DHT actually suppresses estrogen’s effects not by
inhibition of synthesis of estrogen receptor, but by (get ready…big
words coming up) decreasing estrogen-induced RNA transcription at some
point after the actual estrogen receptor binding has occurred. This
means, in much simpler terms, that the estrogen gets to the receptor,
but just doesn’t do its job (1). This means you can take steroids that
convert to estrogen (called aromatizable steroids) and not worry about
that estrogen possibly making you retain water, gain fat, or watch
"Desperate Housewives." Also, this could mean that the antiestrogenic
effect of DHT is mediated by an androgen receptor mediated mechanism.
In fact, DHT has been shown to prevent the estrogen-dependent
augmentation of the progesterone receptor in human breast cancer cells.
And, not to be redundant, but it’s important to remember that virtually
all of the anti-estrogens we use to control gyno and water retention
are also used to treat breast cancer. So, now we know have observed
that androgens are capable of inhibiting both the estrogenic induction
and the ongoing stimulation of PRc synthesis, but have no apparent
effect upon basal concentrations of this receptor. Dihydrotestosterone
(DHT) demonstrates a very high degree of inhibition of estrogen in
human breast cancer cells. (2). But it’s not just DHT that does this;
its metabolites have been shown to inhibit aromatization itself; DHT,
androsterone, and 5alpha-androstandione are all potent inhibitors of
the formation of estrone from androstenedione. In fact, it's so potent
at reducing estrogen that transdermal DHT gel applied to the affected
area has been used to treat gynocomastia (3). DHT is such a potent
anti-estrogen that it been even been used to increase height in
children with short stature, and since it’s been determined that this
increase is not due to GH-mediated effects, it was strongly suggested
that DHT’s anti-estrogenic effects are the mechanism by which it can
increase height (4) Of course, I suspect I don’t need to tell you that
DHT is structurally incapable or converting to estrogen…
So all of this tells us that DHT will certainly have beneficial effects
on keeping our estrogen in check, but what about Masteron? Can it be
used as effectively? Well, let’s take a look at what Masteron actually
is, relative to DHT. But before we can do that, I think a quick
explanation of DHT is in order first. Don’t worry; I’ll make it as
brief and painless as possible.
DHT is actually the result of testosterone interacting with the
5alpha-reductase (5a-R) enzyme. This enzyme is present in the scalp,
prostate, external genitalia, and other places. As far as I can see, it
apparently exists for the sole purpose of converting a steroid with a
double bond between carbon 4 and carbon 5 to one with a single bond
between them, and subsequently adding a hydrogen atom to each carbon.
This process is called (of course) 5alpha-reduction.
So now we know how testosterone becomes Dihydrotestosterone.
everything would be great if this is the only thing that happened to
our good old friend testosterone, because as you may already know, DHT
is a far more potent androgen than testosterone. But, unfortunately,
this is not the end of the story, because DHT is largely deactivated by
the enzyme 3-alpha Hydroxysteroid Dehydrogenase (3bHSD), which is
mainly present in skeletal muscle.
For our purposes here, we’re only going to be concerned with one
particular action of this enzyme. It can either converts a steroid
with a keto group on position 3 of the steroid to one with a hydroxy
group in that position, thus converting DHT is to androstanediol. This
conversion is part of reason DHT alone has not proven to be a very
effective muscle builder, as androstanediol is not going to be very
anabolic at all. If you look off to the left of the following molecular
diagram, and compare it to the one above for DHT, you’ll notice that
the "O" (oxygen) has been replaced with an "HO" (hydrogen + oxygen) at
the third position:
3bHSD is present all over the body (as is 5a-R, for the most part), but
is found in especially high concentrations in the scalp and prostate,
and it’s even possible that its actions on DHT will exacerbate male
pattern baldness in the former tissue. Also,
it’s worth noting that DHT is the androgen responsible for development
of external genitalia. This is most likely the reason that
women experience a temporary clitoral hypertrophy when they use the
often recommended steroids (Primobolan, Anavar, Winstrol,
etc…) in excessive doses. In an interesting aside, I find it really
interesting that the most typical steroids recommended are the
most likely to cause clitoral enlargement and other possible androgenic
effects. But on the bright side, in my experience with female athletes,
that first effect is most welcome...actually, topical DHT can even be
used to treat Microphalia (extremely tiny genitalia) in males (5). This
last fact, if you’ve ever wondered, is the type of information
discussed behind closed doors by of owners and staff of
"private/invite-only" anabolic steroid boards and forums…for obvious
Ok, so now you know what DHT is, where it comes from, what it can do,
and why it’s not a particularly potent anabolic when used
alone. Here’s what Masteron is, relative to its parent compound, DHT.
Masteron is an injectable steroid that is simply the DHT
molecule which has been altered to be 2alpha-Methyl-DHT…you can see
this modification by comparing the DHT molecule above with the
following Masteron one, and paying special attention to the left hand
side again, and the "H3C" modification:
(Masteron, aka Drostanolone Propionate)
This 2-alpha-methyl alteration makes it much more potent anabolic,
although it’s still only about 60% as anabolic as testosterone and a
quarter as androgenic. I’m going to speculate that these ratings make
it not the most potent anabolic in the world, but its anti estrogenic
effects plus its ability to increase aggression make it a very nice
pre-contest addition. This is also where we get the absurd rumor that
Masteron won’t do anything for you unless you’re already at a very low
body-fat percentage. This is not true at all. No matter what body-fat
percentage you’re at going to get a nice anti-estrogenic effect from
Masteron, as well as some nice aggression and strength in the gym - the
former and latter are both known as "non-genomic" effects, and are a
result of the strong Central Nervous System stimulatory effects of
Masteron, which is very common with DHT derived steroids. Basically, if
you’re fat, and you take something that increases aggression and has
anti-estrogenic effects (Halotestin and Arimidex, lets say), you
wouldn’t expect to get huge and ripped. It’s the same thing with
Masteron. Now, what if you add in Arimidex and Halotestin to a
pre-contest cycle, you’ll get harder and look better. That’s exactly
what’ll happen if you add Masteron into a Pre-contest cycle. It’s not
that you have to be at some random body-fat percentage to get results
from it, but you’ll need to be at that lower body-fat percentage to
"see" those results. Again, if you’re fat and take Halo and Arimidex,
you aren’t going to look much
better…think of Masteron in similar terms, but it won’t work as well
for aggression as Halotestin, and won’t be as good for combating
estrogen as Arimidex. Gauged against either one of them alone, Masteron
will likely make you look much harder and lift more weight. But if you
are looking to do a low dosage cycle with a minimal amount of compounds
in it, a simple Testosterone (propionate) and Masteron cycle may be
exactly what you are looking for. On a personal note, that is a cycle
that I use very
frequently, at about 100mgs of each, shot every other day.
But has Masteron actually lived up to my claims for being an
anti-estrogen? Yes. From 1968 to 1972, a decent sized study was
conducted on Masteron, in a group of premenopausal women with breast
cancer. About a third responded well to Masteron (6). This is because
of its anti-estrogenic effects, clearly- though it doesn’t perform as
well as Arimidex, Letrozole, or Aromasin. If
you’re not running huge amounts of aromatizable steroids, this is a
very good choice to add into your cycle. If you’re
doing large amounts of those compounds, then you need to use a
traditional anti-estrogen as your ancillary compound of choice. But if
you’re running well under a gram of aromatizable steroids, Masteron
will likely be all the anti-estrogen you need. This number
comes from my person experience, as well as others I’ve interviewed.
Now, as a bit of an addendum, I’d like to address the use of Masteron
in women. Lets get this straight: Masteron was developed for
women. Okay? Got me? If you’ ve been paying attention up to this point,
you already know that Masteron is intended for females and
is derived from the same root (DHT) as most other steroids commonly
used and recommended for female athletes (Primobolan, Anavar, Winstrol,
etc…are all derived from DHT). And, another shocking fact is that
Masteron has a lower androgenic rating than almost every other commonly
recommended steroid used by female athletes. Anavar has a rating of 24
compared to oral testosterone and Masteron has a rating of 25 compared
to testosterone, expressed as a percent (so yes that means 24% and 25%
Basically, Masteron works as a hormonal therapy for breast cancer and
has been shown to be a useful and safe agent for females of all age
groups, even though it may appear to be less effective then other
possible therapies in postmenopausal patients (6). It is, therefore,
very safe for women. Masteron is certainly no less safe than Anavar or
Primobolan for women, as long as it’s used with something
resembling a degree of respect and intelligence.
My recommendations for female use of this compound would be to start
between 10-25mgs every third day, and increase dosages from there if no
side effects are experienced. At those dosages, I suspect no side
effects would be experienced, and I’d be comfortable saying
none will be experienced up through 20mgs, injected every other day.
So there you have it. A totally new way to look at an old friend-
Masteron- it’s useful as an anti-estrogen as well as an
anabolic, and can certainly be safely used by both Men as well as women.
Successful percutaneous dihydrotestosterone treatment of gynecomastia
occurring during highly active antiretroviral therapy: four cases and a
review of the literature. Clin Infect Dis. 2001 Sep 15;33(6):891-3.
Epub 2001 Aug 10.
Hormonal therapy of breast cancer with special reference to Masteron
therapy. Bennett MB, Helman P, Palmer P PMID: 1242823).
Targex - The Ultimate Topical Fat Burner!
Glycyrrhetinic acid is the primary ingredient in Targex. It is actually
found in licorice, and when applied to the skin, can reduce the
thickness of subcutaneous fat. In this case, the mechanism of action is
mediated by the catabolic hormone known as cortisol. This
hormone is involved in both the distribution as well as the deposition
of fat.Cortisol, in turn is regulated by the activity of an enzyme
which Glycyrrhetinic acid (the active ingredient in Targex) blocks.
This reduces the ability of cortisol to regulate fat cells, resulting
in an overall loss of bodyfat to the area the cream is applied to.
In a clinical study, the effect of topical Glycyrrhetinic cream was
evaluated. In this study, measurements of thigh fat, before and after 1
month of treatment with the cream were taken. In both areas
(circumference and the fat layer thickness) the thighs receiving the
cream had significantly lower levels of fat.
Hence, this type of cream would appear to be a potent lipolytic (fat
burning) agent. Also, it is important to note
that this compound is very safe, as the full study mentioned previously
states that that there was absolutely no change in
plasma cortisol, blood pressure, or aldosterone.
The primary ingredient in that study (Glycyrrhetinic acid) is also the
primary ingredient found in Targex; this is actually the brand name of
the original version of these types of products. Typically it is
applied to specific sites, to produce localized fat-loss wherever it is
applied. The typical Targex user has experienced significant reductions
in body fat when it was applied to site-specific areas, and was used in
combination with a proper diet, training, and cardio.
Glycyrrhetinic acid, the active principle of licorice, can reduce the
thickness of subcutaneous thigh fat through topical application.
Armanini D, Nacamulli D, Francini-Pesenti F, Battagin G, Ragazzi E,
Department of Medical and Surgical Sciences-Endocrinology, University
of Padua, Via Ospedale 105, 35100 Padua, Italy.
Cortisol is involved in the distribution and deposition of fat, and its
action is regulated by the activity of 11beta-hydroxysteroid
dehydrogenase. Glycyrrhetinic acid, the active principle of licorice
root, blocks 11beta-hydroxysteroid dehydrogenase type 1, thus reducing
the availability of cortisol at the level of adipocytes. We evaluated
the effect of topical application of a cream containing glycyrrhetinic
acid in the thickness of fat at the level of the thigh. Eighteen
healthy women (age range 20-33 years) with normal BMI were randomly
allocated to treatment, at the level of the dominant thigh, with a
cream containing 2.5% glycyrrhetinic acid (n=9) or with a placebo cream
containing the excipients alone (n=9). Before and after 1 month of
treatment both the circumference and the thickness of the superficial
fat layer of the thighs (by ultrasound analysis) were measured. The
circumference and the thickness of the superficial fat layer were
significantly reduced in comparison to the controlateral untreated
thigh and to control subjects treated with the placebo cream. No
changes were observed in blood pressure, plasma renin activity, plasma
aldosterone or cortisol. The effect of glycyrrhetinic acid on the
thickness of subcutaneous fat was likely related to a block of
11beta-hydroxysteroid dehydrogenase type 1 at the level of fat cells;
therefore, glycyrrhetinic acid could be effectively used in the
reduction of unwanted local fat accumulation.
If you read the original abstract you will note that ...
"No changes were observed in blood pressure, plasma renin activity,
plasma aldosterone or cortisol",
There was no change in plasma cortisol; cortisol was lowered only in
the fat cells, this is telling us that little if any of the compound
was absorbed systemically or we would see a drop in whole body, i.e.
PLEASE SHAKE BOTTLE BEFORE USE
Apply 1 to 2 pumps ( one pump = one CC ) applied TWICE daily in desired
areas and rub vigorously until almost vanished into the skin of area
applied, though this lotion is greasy of nature is necessary for it to
The area must be clean and free of hair.
This lotion will NOT stain clothes.
There are some studies that claim Licorice extract may increase levels
of the hormone adolsterone if there is a systemic uptake BUT our unique
TDS formual DOES NOT allow this to happen and TargeX works on the site
110% SAFE AND NON-TOXIC!
HELPFUL INFORMATION ON HOW TO USE TARGEX ...
So I have some problem areas where I store more fat or simply I got
some flabby stubborn fat like for example in my oblique , inner thighs
and lower pecs , my diet and cardio has been in check but still I need
Well, TargeX will not shift your body into a calorie burning machine
like other compound do ie. Clenbuterol , T3 , Ephedra , etc...but BY
BLOCKING CORTISOL AT THE CELLULAR LEVEL AND PREVENT IT IT FROM STORING
FAT WHEN THIS HORMONE "CORTISOL" IS AT PEAK ( BEFORE RETIRING, UPON
AWAKENING AND POST-WORKOUT ) Of course add a good diet and cardio and
you'll see visible results in the areas applied.
***PLEASE NOTE NOT ALL RESPOND IN THE SAME TIMELY MANNER AS SOME GET
RESULTS IN ONLY 2 OR 3 DAYS AFTER INTIAL USE AND OTHERS IT MAY TAKE
Ok, so I got a 4oz bottle of TargeX and it contains 120cc of 40mgs of
G.Acid 98% purity per CC enough for a 2 week cycle
which is what i recommend as our wonderful bodies will fight it, after
all cortisol hormone is a necessary evil so cycling 2 weeks on - 2
weeks off one area at a time is best !
Patience and common sense is needed here, please allow me to explain
why and how:
I want to decrease the stubborn fat I got on my oblique both left and
right so :
Before I go to bed I apply 2ccs of TargeX on my right oblique then rub
vigorously until vanished ( I like using a hair
blowdrier in Med tempetature to help dry area faster )
Then I apply another 2ccs on my left oblique and rub again until
vanished and absorbed.
Next day when I wake up I repeat procedure again.
***Make sure the area you apply TargeX to is free of hair and is clean.
Using TargeX this way is safe and ONE 4oz bottle should be enough for a
2 week cycle
and may have some extra.
Ok, now you see some improvement in the areas you applied to but you
need more treatment so you start again another 2 week cycle
after at least 1 week off TargeX, if you are happy with your results
then move on to another area you desired to reduce stubborn
subcutaneous fat. This time it may the lower pecs so you do exactly as
you did the first time when you applied to your oblique.
***Please keep in mind this is not miracle drug ( well almost ) and
that the area you worked first may need some more treatment but you
need to do your part as far as diet and cardio, this is true in all
products that help reduce body fat, do not tell me is not true cause I
tried them all and when my diet was sloppy I made no progress.
***Work one area at a time, I mean for example again oblique ( left and
right ), lower pecs ( left and right ) inner thighs ( left and right)
and so on
***I recommend using an over the counter dietary supplement that
reduces excess estrogen , is my belief that the presence of excess
estrogen makes that stubborn fat hang on to those problem areas. This
is just my own opinion based on personal experiences.
So there you have it. So the next time you decide to ‘bulk’
have much more options than you previously thought.
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